“We do not believe any group of men adequate enough or wise enough to operate without scrutiny or without criticism. We know that the only way to avoid error is to detect it, that the only way to detect it is to be free to inquire. We know that in secrecy error undetected will flourish and subvert.” –J. Robert Oppenheimer
TL;DR – Watch this sucker while ignoring the myopic neo-Bolshevik “refugee” propaganda, but instead keeping in mind the current COVID/WEF insanity, to get a grip on the incipient climate-excused holocaust of the depopulationists, which is the fault of SOCIALISM, same as the soon-to-be-dwarfed “Holocaust” of the 1930s and 1940s.
OK – just a bit more detail…..
Don’t watch the Ken Burns Holocaust special for any of the slanted, leftist, socialist messaging stapled onto the historical facts in a DNC-approved and ADL-edited way.
Watch this PBS special while subversively keeping in mind the new socialist, depopulationist, eco-nuttery holocaust which now exceeds 6 million COVID dead and will soon DWARF the old “Holocaust” of so much hypocritical and self-centered angst.
Calmly stroke Suspicious Cat, as the Holocaust is falsely linked to rejection of current “asylum seekers” in a face-palmingly stereotypical and Trump-hating way.
Watch this PBS special while “rebelliously” remembering that RIGHT NOW we are in a DESIGNED CRASH of the world economy, which will kill BILLIONS of innocent people if not stopped – and which is largely driven with the eager participation of globalist and socialist “Jazis” – who are no less evil than the Nazis of old – self-righteous in their “ESG/DEI” zeal, with their vain self-assurance that they and they alone know the answers which must be forced upon the world.
Watch this special remembering Andrew Cuomo, Anthony Fauci, Ralph Baric, Joe Biden, WEF, Klaus Schwab, Pfizer, CCP and Albert Bourla, and keeping close in mind, the NEW holocaust which has already killed over 6 million worldwide by the virus ALONE, and is already creating even-more-massive excesses in mortality in every country, in unacknowledged proportion to acceptance of the “vaccines”.
Watch this PBS propaganda special while remembering how the US and China co-created biological “gain of function” which literally DWARFS the mere chemical nastiness of a bug spray, “Zyklon B”, itself more comparable to Fauci’s murderous remdesivir, or the midazolam they apparently used for “COVID euthanasia” in the UK.
Watch this special while remembering the millions who died around the world from both virus and vaccines, including tens of thousands of Israelis, as secular Jewish leaders condemned their own and didn’t lift a finger, other than to censor Zev Zelenko, and to suppress their own report on the dangers of the vaccines they INVESTED IN.
Watch this special in strict analogy of the THEN unbelievability of the “final solution”, to the NOW unbelievability of “depopulation”.
Realize that the problem isn’t antisemitism or “refugee acceptance” – it’s SOCIALISM.
SOCIALISM KILLS – no matter which brand.
Representative Alexandria Ocasio-Cortez (D-NY) and Senator Ed Markey (D-MA) were joined by Democratic lawmakers from both the House and Senate on February 7, 2019, to introduce Green New Deal legislation.
Oh, hell. I said enough already. Do I really need to explain this?
If so, I’ll do it in the comments.
But the bottom line is that this special has a lot of great facts, which – if you are prepared to see them in the context of current events – are quite convincing that we ARE in the late beginning of yet another socialist holocaust.
If you are saying BAD THINGS about the wonderful coronavirus vaccines with safe and effective mRNA technology, which have never killed or harmed anybody, then you need a REFRESHER COURSE in the RULES OF JAB CLUB.
#1 – The first rule of Jab Club is, you do not talk about Jab Club.
#2 – The second rule of Jab Club is, you DO NOT talk about Jab Club.
#3 – If someone dies or gets injured, it wasn’t the jab.
#4 – Two jabs to a vaccination.
#5 – One jab to a booster.
#6 – No admission of harm, no metrics except antibodies.
#7 – Jabs will go on as long as they have to.
#8 – If this is your first night in Jab Club, you have to get jabbed.
This kind of mistake is completely unacceptable. Although Mr. Bongino did not violate the first rule of Jab Club, which is to talk about Jab Club, he came very close. It’s clear that people are not following the rules of Jab Club, hence the need for this refresher.
We are providing the following examples for your education and sense of well-being.
#6 – No admission of harm, no metrics except antibodies.
You can’t say things that give people ideas.
The disease can’t hurt rabbits, because the jabs can’t hurt humans. Get it? Understand spike protein political vulnerability! Loose lips cause slips! Corona viruses are potentially bad diseases, that limit human life, but if we talk about any specifics, people will figure out that the jabs do the same thing, only worse, and that we’re making money on THEIR DEATHS.
Why, people might even figure out that we made the damn virus more deadly on purpose, to help depopulate the planet of all these annoying plebes!
Enough already! Wise up!
Example 2
EUROPE — "The country with the lowest vaccination rate has the lowest excess mortality rate and the country with the highest vaccination rate has the highest excess mortality." pic.twitter.com/qslvKukPUq
#3 – If someone dies or gets injured, it wasn’t the jab.
It wasn’t the jabs. EVER.
The FAUCI WALL admits no blame. SUCK IT UP, PEOPLE!
Strike back against this one with “correlation does not prove causation”, said with a tone of certainty and finality. Remember: Follow the science means don’t question the jabs!
This one is a complete travesty, from talking about “sudden death” and individual athletes, to talking about things like overall mortality rates AT ALL. But it gets worse.
“Denial” is actually mentioned!
“DENIAL” is a code word for JAB CLUB!
REMEMBER!
#1 – The first rule of Jab Club is, you do not talk about Jab Club.
#2 – The second rule of Jab Club is, you DO NOT talk about Jab Club.
Check out the video – it’s a superb Jab Club TRAINING VIDEO!
https://youtu.be/61hmUuSewSY
See how that works? Just like back in Germany during the 1940s. People knew everything was OK for the Jews. Hitler was taking care of them, just as he promised. All those stories about maltreatment were misinformation – just like the misinformation we are experiencing now about the vaccines.
Yes, children are experiencing slightly more myocarditis these days, and government scientists are hard at work, figuring out why this might be. There are many possible candidates, including anxiety about gender, concern about insurrectionists, air pollution, and especially climate change. But children should not worry about disease – myocarditis is easily treated.
You may see graphs like this one.
If you see something, say something. This graph and others like it are likely to be misinformation, and should be reported to authorities, just like Jewish propaganda was successfully reported to the appropriate authorities around 80 years ago, in a similar fashion.
There are NO WORRIES when you follow the rules of Jab Club!
We hope that you fully understand the rules of our wonderful JAB CLUB that make sure we’re all safe, happy, and in excellent health.
Now – is your FIGHT CARD – whoops – I mean your VACCINE PASSPORT current?
If not, GET A BOOSTER! But just one! Remember Rule #5:
#5 – One jab to a booster.
The reason for Rule #4 (two jabs to a vaccination) is that it takes two shots of the deadlier Wuhan variant spike protein to seriously injure most people, and in particular for the second shot to create a bad reaction to the first shot.
The reason for Rule #5 (one jab to a booster) is to let each booster have enough time to kill people. Most people who are going to die, die about five months after the shots. Start bunching up the boosters, and it’s likely that people will die shortly after a booster, which makes Rules #1 and #2 more difficult to enforce.
And THOSE TWO are the most important rules!
#1 – The first rule of Jab Club is, you do not talk about Jab Club.
#2 – The second rule of Jab Club is, you DO NOT talk about Jab Club.
KNOW THE RULES, PEOPLE!
Science
Dr. Anthony Fauci
W
“What are you doing out of bed? I think you need some more REMDESIVIR.”
PS…..
Hat Tip slowcreekno for coming up with the concept of “Jab Club” while answering my question about breaking through the “we can’t talk about the evil jab” wall of denial.
Why would we mass treat a virus with a drug which forces the virus to mutate, when mutation is how the virus creates new variants that reinfect the vaccinated?
Before I explain the title contradiction, let me start with an admission.
Most of my life, I have been very friendly with the pharmaceutical industry. I have eloquently defended Big Pharma, the FDA, “government and corporate medical science”, and all those things that the Biden administration so earnestly defends now.
I even got an award sponsored by one of those Big Pharma companies – which is not to say much, because they give out a LOT of them. In fact, the grooming of young scientists to revere Big Pharma, is no different from the grooming of doctors (and now medical bureaucrats, who know less “talk-back” science) to promote and prescribe their products.
If you go back and look through my posts here, you will see that my thinking about Big Pharma has only evolved slowly from starry-eyed hope and blissful faith. I was quite earnest in my wishes that some of their new products might be better than doctor-discovered, repurposed, off-label drugs like hydroxychloroquine and ivermectin.
What I would NOT do, was deny the obvious effectiveness of those cheap, plentiful, and SAFE doctor-discovered drugs.
If the world was against HCQ, then “Lupus contra mundum” (Wolf against the world).
Why so? Because the DATA on these two drugs killing virus and preventing death was so alarmingly GOOD. You just have to be HONEST and INDEPENDENT to see it. Then, you just ask WHY. And the answers came.
It was BEAUTIFUL. It was SCIENCE.
Even when it was ugly. Like the Lancetgate Effect.
I’m a DATA GUY. I know WHICH data matters and WHICH data doesn’t. I can SEE THROUGH CURVES like a horny guy next to a woman in bed in the dark, seeing her under the sheets. With DATA, I can see through walls. I can see around corners. I have escaped death many times by seeing what nobody else saw.
It’s a gift from GOD, and I don’t waste it.
I really WANTED remdesivir to work, but then I saw the numbers. I could not unsee them. I was forced to admit that the drug DID not work, and COULD not work, in large part because it was being administered too late.
Antivirals work best EARLY, when they have an overwhelming numerical advantage – which is very hard to obtain over an EXPONENTIAL ENEMY. But if you administer early, even ATYPICAL antivirals like hydroxychloroquine and ivermectin, in proper ANTIVIRAL doses, have a chance.
Remdesivir is fairly toxic stuff, and when administered too late, when the virus is long gone, it kills its victims in a way surprisingly similar to what late-stage COVID does, by kidney failure, and then pulmonary dysfunction which looks like pneumonia. So if you administer remdesivir to dying COVID patients, it may not do THEM any good, but it will make YOU a whole lot of money on their deaths, which are thus ENSURED. And YOU won’t get caught doing it, because it all looks like COVID.
There will be justice for Veronica Wolski, because we will DEMAND IT. And until there IS justice, we will drag the CRIMES of Anthony Fauci and Gilead “Pharmaceuticals” and their SLEAZY ASSOCIATES thorough the headlines, over and over, until people SPIT IN THEIR PATH as they walk down the streets. So where do we begin? …
Remdesivir goes really well with murderous vents and no prior therapeutics, and NO, NO, NO ivermectin allowed, which – DO TELL – is exactly how the Stalinist Biden-Obama-Harris administration and its CHINAZI allies kill off us pesky American seniors.
But that’s getting a little ahead of things. We’ll come back to remdesivir.
First – molnupiravir.
Molnupiravir was once called EIDD-2801, back when it was more of a hope and a dream.
I had high hopes for molnupiravir back then. I had hoped it would be a significantly better antiviral than hydroxychloroquine and ivermectin, both of which are antiparasitics first, and antivirals second – and at that, only by a bit of luck. But that LUCK can SAVE YOUR LIFE.
That was back when I didn’t realize how diabolical the people who CONTROL Big Pharma really are – that they would SHIT on a lucky, life-saving break, just for money.
As it turns out, molnupiravir is roughly as good as the cheaper drugs, but definitely not as safe.
Nevertheless, molnupiravir is NEW, it’s PATENTABLE, and it’s a MONEYMAKER. The system is RIGGED, and thus we are DENIED the cheaper, safer drugs, so that our money will fund expensive drug research.
Whatever. That is just the way things are. I didn’t know that, when I was a student. I didn’t realize that the system was actually corrupt. Although the system probably wasn’t as bad back then, either.
Chinese communist ethics have filtered into America, and they have not done Big Pharma any good.
Would I take molnupiravir? Maybe. If I had to pick ONE, it would probably be ivermectin. Second choice, hydroxychloroquine. Third, molnupiravir. I don’t think I would take remdesivir next – I’d probably try acyclovir. That stuff really WORKED for my shingles – TWICE. It might not work on a coronavirus, but at least it wouldn’t kill my kidneys.
Now that you know how I feel about the drugs, let’s talk about WHY I feel that way. But in a roundabout and very telling manner.
Here is a synthesis of molnupiravir from cytidine – the molecule that it mimics in order to kill RNA viruses, including SARS-CoV-2.
If you look at the molecular structure of molnupiravir above, on the right, you will see two rings. The pentagonal ring with an “O” (oxygen) is a SUGAR ring, and the hexagonal ring with two “N” (nitrogen) atoms is a BASE ring.
Together, those two rings are ALMOST a nucleoside – a component of RNA – called cytidine, shown above on the LEFT, or below.
The only real differences between molnupiravir and cytidine, as shown, are the tail on the left of molnupiravir, hanging off the left-hand O group (and which really only helps the delivery of the drug), and more importantly, that extra “OH” group, hanging off the right-hand NH group of the molnupiravir molecule, in the diagram above.
Add that OH group to cytidine, and you have N4-hydroxycytidine (NHC) – the “real” drug being administered, also known as EIDD-1931. Add that little ester tail on the left, to make a nice orally active and bioavailable “prodrug” of NHC, and you have molnupiravir, or EIDD-2801.
That OH group totally screws things up. It’s absolutely AMAZING what that does to the genetic machinery of the virus, inside YOU.
FAKE cytidine, like FAKE NEWS, kills.
There is a great but still fairly technical explanation of how molnupiravir works that was published in Nature, called “Molnupiravir: Coding for Catastrophe“. You can download a PDF of the article HERE.
The abstract is very useful:
Molnupiravir, a wide-spectrum antiviral that is currently in phase 2/3 clinical trials for the treatment of COVID-19, is proposed to inhibit viral replication by a mechanism known as ‘lethal mutagenesis’. Two recently published studies reveal the biochemical and structural bases of how molnupiravir disrupts the fidelity of SARS-CoV-2 genome replication and prevents viral propagation by fostering error accumulation in a process referred to as ‘error catastrophe’.
I used part of one graphic from the paper for the feature image of this article. That graphic shows crude, flattened structures of both molnupiravir, and the fully phosphorylated fake nucleotide that gets incorporated into the virus RNA, which is called molnupiravir triphosphate, or MTP.
Technically, it’s really not molnupiravir any more, after that prodrug ester gets replaced by a triphosphate unit – it should really be called N4-hydroxycytidine triphosphate. But that pickiness is confusing – MTP is still very true in spirit, and that’s FINE with us big picture types.
Now – THIS is where it all happens. This is where THINGS GO WRONG, and the drug starts to work.
That OH group hanging off the NH of molnupiravir CHANGES the nature of the nitrogen atom to which it is attached, and in a BIG way. Suddenly, the little hydrogen atom that is attached to that nitrogen, would almost rather be located on the OTHER nitrogen in the ring, instead of staying where it is, on the sideshain nitrogen, next to OH. In fact, that hydrogen atom almost stops caring which place it stays. This is a phenomenon called tautomerism. It’s a molecule that can exist in two forms.
One little proton. It’s now happy either way.
But RNA? It ain’t happy.
So what happens, is MTP goes into RNA where CTP should go. And once M is in there where C should be, M can’t make up its mind where that little proton should go. If the machinery sees M with the hydrogen where C would keep it, the machinery does the right thing, and M just gets treated like C. No mutation. But if the hydrogen is in the other place, the machinery thinks M is actually U, and a mutation occurs.
You can see that in this next diagram, where the “hydroxylamine” (-NHOH) form binds correctly with GTP, but the “oxime” form (=NOH) binds INCORRECTLY with ATP.
In the next graphic, you can see how M gets incorporated for C, and starts to cause problems by leading to U instead of C. The events shown in the graphic follow a sequence I’ll try to describe.
If you can’t follow it, don’t worry. This stuff is always confusing when you track the changes.
Starting from the top, below……
one ringer M is already present (top strand), while M competes with C to match the next G (two choices shown waiting)
the second ringer M goes in on the bottom strand, to match the G, where C should have gone
the second ringer M (now on top, follow UACGM from left) is then matched with a new A (WRONG) on the bottom, instead of a G (two choices shown). You can also see (and this is very complicated) that the first ringer M was matched with a G (now shown on top), and that G has already matched up to ANOTHER (third) ringer M, now on the bottom strand in the third subgraphic.
the strand with incorrect A (follow UMAA from right to left on bottom, now on TOP, right to left) is then matched with a U on the second A, completing the screw-up from C to U
the net effect, bottom strand, is that UACG[C] (top of diagram, what should have happened) became UACG[U] (bottom of diagram, what did happen)
One can look at this whole process as N4-Hydroxycytidine (M) cutting in line where C was supposed to go, and then handing things off to the WRONG base, so that C gets replaced by U.
Complicated, isn’t it? But THAT is how mutations are PROMOTED by this drug, and they are KEY to how it works. There is an AVALANCHE of mutations that kills the virus. The whole idea is that the DRUG makes the virus mutate too much, too fast, into non-viable forms, and it just dies – or at least enough for your immune system to take over and WIN the fight. The virus CRASHES because of the drug. Meanwhile, the body mounts a defense.
You can read the rest of the article if you want, and get some sense of the complexity of considerations as to whether this makes a good drug or not for the individual.
There IS a legitimate question of whether screwing up the RNA of the virus, might also lead to screw ups in the host – either in RNA or DNA, leading to things like birth defects, cancer, adverse events during therapy, etc.
That concern is nicely summarized in a Zero Hedge article:
“Proceed With Caution At Your Own Peril” – Merck’s COVID ‘Super Drug’ Poses Serious Health Risks, Scientists Warn
Now, I’m not really interested – for the purposes of this article – in the question of whether or not there are INDIVIDUAL dangers posed by molnupiravir, due to either mutations of the host, OR the forcing of mutation of the virus in that host.
There are excellent reasons to believe, that just like vaccines don’t really pose INDIVIDUAL risks through mutation of the virus in any particular victim, there is no significant individual risk from mutations of the virus due to a mutagenic drug.
HOWEVER, that’s not my concern.
My concern is related to Dr. Geert Vanden Bossche’s concern about mass vaccination during a pandemic. He differentiates between the idea of a vaccine being good for an individual, and that vaccine being good public policy for humanity as a whole, ultimately including that individual.
Geert’s concern is that a virus AS A WHOLE – as a global population – as almost an ecosystem – will evolve due to pressure from a non-sterilizing vaccine, to create new strains that will resist the vaccine. Thus, while the vaccine may benefit an individual in the short term, it ultimately does NOT benefit the sum of all individuals, who will ALL suffer from the mutated virus, which would not have happened, absent the specific evolutionary pressure of the vaccine.
If Geert is right, it’s not just stupid to “vaccinate ourselves into trouble” – it’s downright EVIL.
We have already seen Geert’s prediction apparently (wait for it) fulfilled with the delta strain of SARS-CoV-2, which basically ignores vaccines against “wild type” Wuhan coronavirus.
But again, that is not STRICTLY my concern.
Then what IS my concern?
Original predictions, based on the mutation of the original Wuhan coronavirus, were that the virus was genetically contained – that it was not mutating into significantly different forms requiring changes in the vaccine. And yet, something seems to have CHANGED that. The early predictions could have been WRONG, but they could also have been UNDERMINED. And they could have been undermined by the same terrible logic of “we have to pass it to see what’s in it”, or “we have to try to MAKE the virus catch in human cells, to see if it CAN catch in human cells”.
You see what I mean? There could be “dishonest science” and other such “skulduggery” here, just like we have seen with LIARS like Fauci, Baric, Tedros, and China.
My concern is that in Geert Vanden Bossche’s scenario, which I have described as “coronavirus variant whack-a-mole”, it will only be made WORSE by drugs which encourage the mutation of the virus.
In other words, mass vaccination into a pandemic with “leaky” vaccines is bad, but to do so while chemically promoting the mutation of the virus is even worse.
Thus, not only is it CONTRADICTORY to vaccinate in such a scenario – it is EVEN MORE contradictory to promote mutation in such a scenario.
And – worse than THAT – it appears that we have ALREADY BEEN DOING IT – with remdesivir.
Remdesivir is notable as being an antiviral which is generally being given to patients, with no hope of it actually working, long after the SARS-CoV-2 virus has done its dirty work, and those patients are ACTUALLY dying of a cytokine storm. These patients may still be producing and shedding some virus, but the sum of all studies is rather definitive at this point – remdesivir does little except LENGTHEN the stay of patients in the hospital.
Well, what are those patients doing there, staying too long in the hospital?
One strong possibility is that these dying patients are creating mutants and variants. The following paper shows what happens to SARS-CoV-2 virus when confronted in vitro with remdesivir – and it is basically what I am predicting will happen with molnupiravir.
In vitro evolution of Remdesivir resistance reveals genome plasticity of SARS-CoV-2
Remdesivir (RDV) is used widely for COVID-19 patients despite varying results in recent clinical trials. Here, we show how serially passaging SARS-CoV-2 in vitro in the presence of RDV selected for drug-resistant viral populations. We determined that the E802D mutation in the RNA-dependent RNA polymerase was sufficient to confer decreased RDV sensitivity without affecting viral fitness. Analysis of more than 200,000 sequences of globally circulating SARS-CoV-2 variants show no evidence of widespread transmission of RDV-resistant mutants. Surprisingly, we also observed changes in the Spike (i.e., H69 E484, N501, H655) corresponding to mutations identified in emerging SARS-CoV-2 variants indicating that they can arise in vitro in the absence of immune selection. This study illustrates SARS-CoV-2 genome plasticity and offers new perspectives on surveillance of viral variants.
Now this is moderately straightforward, but the big picture is not apparent, because the authors know they are playing with dynamite, so I’m going to restate what they found in more direct language.
Bottom line up front, they basically found evidence that remdesivir does exactly what I’m thinking molnupiravir will do – which is to promote mutation per se, including into “variants of concern”, independently of drug resistance evolutionary considerations, which makes tons of sense.
A mutagenic drug (or rather a drug which works on the principle of mutagenesis) creates mutations with high frequency on a large scale, without the need for evolution to strongly amplify rare beneficial mutations. But at the same time we don’t see – in the wild – any evolution of resistance to remdesivir (RDV). The paper spells this out.
So let’s look at what the study found:
“in vitro with omnipresent RDV” – we see both appearance of variants of concern AND resistance to RDV
“in vivo with late-stage RDV” – we see appearance of variants of concern but NO resistance to RDV
[ The second is a bit of a joke – I’m talking about what we see in the wild globally – no RDV resistance. ]
How can this be rationalized?
In the in vitro case, resistance to RDV is a NECESSITY forced upon the virus. All mutations must persist under omnipresent high concentrations of RDV, so this is a pressure that cannot be worked around or escaped from. Yes, RDV benefits the virus by assisting mutation, despite doing it “too much”, which forces resistance to occur. And what IS the resistance? It is for the virus to continue propagating, both unhindered by RDV yet also assisted by RDV. So, essentially, SARS-CoV-2 and RDV negotiate to the point where the “benefits” of RDV to speed up mutation don’t diminish the viability of the virus. The virus learns to USE the ringer nucleoside M to mutate faster, without dying from it. Thus, we see evolution of traits that have benefited SARS-CoV-2 in the wild, plus evolution of a trait of adaptation to RDV.
In the in vivo case, in a Petri dish called “planet Earth”, resistance to RDV is NOT a necessity. The virus has plenty of hosts who are not using it, so it negotiates more strongly to a better deal. It takes all the mutations it can get from RDV, but it does NOT accept the need to mutate to adapt to RDV. THAT particular mutation is unnecessary for most of the virus, so it is not forced to cut that deal.
Bottom line question: Does RDV in the wild speed up mutation?
My answer: I would bet money on it. It appears to do so in the lab.
And if I’m right, enhancement of mutation should happen even more strongly for molnupiravir, which has a more clearly mutagenic mechanism of action than remdesivir.
The authors simply refer to the plasticity of the VIRUS, because woe unto them if they talked about a Big Pharma drug being a promoter of viral plasticity-COUGH-mutation. But that is exactly what the in vitro results mean here. They were able to generate the “variants of concern” in the lab, using exposure to remdesivir.
They went looking for mutations for resistance to remdesivir, and they not only found one of those – they found MORE mutations, including ones matching “variants of concern”.
WHY?
Well, let’s go back to the original point:
Why would we mass treat a virus with a drug which forces the virus to mutate, when mutation is how the virus creates new variants that reinfect the vaccinated?
In my opinion, it is REASONABLE to expect that any drug which operates as a “ringer” nucleoside – as BOTH remdesivir AND molnupiravir do – is going to cause SOME level of genetic errors – a.k.a. mutations – as a consequence. You can dress up pro and con arguments in fancy language, but scientific common sense points one to the likelihood that a fake nucleoside will operate to some extent, if not to its main extent, as BAD DATA in the tape of life. And THAT means MUTATIONS.
And if remdesivir was doing it, then molnupiravir should do it on STEROIDS.
And I am NOT going to let Fauci explain his way out of this one by any kind of hand-waving, or Shifty-Schiff experiments like Lancetgate.
So where does this go?
I was having a lot of trouble figuring out why the push for remdesivir made sense to a particular PART of the corrupt forces behind the Plandemic.
Remember – AND logic.
In any rally of a giant societal “conspiracy”, which can be as big as:
“Let’s all go to the New World for each of our own reasons! It’s OURS!”
“The Islamic world attacked our towers! Let’s DO SOMETHING!”
“The other people are INSURRECTIONISTS! Arrest them!”
“White supremacists! Take away their rights!”
“It’s airborne Ebola! Civil rights out the window! We’re all gonna die!”
…..there is always a REASON for every aspect and for every beneficiary, but they’re usually quite different reasons, specific to the individual or group, and thus profoundly motivating.
In other words, these are “conspiracies of fortune”, in which MOST buy in not in an illegal way, but in either an immoral, amoral, or self-deceiving way. Some truly guilty ones secretly initiate the money-grab, and everybody else goes along, making true justice impossible.
It’s a great scam. It happens for ALL of the reasons – not just any one of them.
Still, in that context, things tend to make sense, but generally after the fact.
The advancement of remdesivir just didn’t make SENSE. More than that, its whole terrible history was wrapped up with the liar Anthony Fauci.
But if you back up even further – a useful tactic when things don’t make sense – one comes to the realization that many things about antivirals just don’t make sense.
we have good safe ones that “they” seem to hate now, upon their “discovery”
those drugs were never promoted or studied properly, IMO
the excuses for not vigorously pursuing the class of drugs BEFORE, ring VERY hollow NOW
the main class of “allowed” antivirals (ringer nucleosides) seems fundamentally flawed
the fundamental flaw (that we are using genetic error as a “cure”) is never acknowledged
the fact that we have to cure diseases that never had to exist, like SARS-CoV-2, fails to outrage any of the people in charge, who pushed these Frankenstein gain-of-function experiments to begin with
there is a bizarre fixation of vaccines as the only allowed solution to viral disease
genetic vaccines are pushed, when antigen vaccines are obviously fundamentally safer
genetic antivirals are pushed, when other categories are obviously both safer AND more effective
The LAST points seem to show some commonality, both in leading toward the massive money pit of gene therapy, and in relating to Anthony Fauci.
And THAT is where things start to make sense. The POLITICAL aspects of this. The installation of World Government, their holy grail.
Fauci, Baric, Daszak, Rick Bright, and Hillary Clinton all know what is actually going on – I am convinced of that. They are all knowledgeable, more than others, in the true agenda and schedule of the “Plandemic”, including the POLITICAL GOALS. They understand both the SCAM and the NOBLE (lying) PURPOSE.
I am convinced that VARIANTS are a key construct in the giant grift of COVID. The whole plan has to keep going, by ginning up more COVID as needed, but it also has to look NATURAL, so that nobody finally decides to send about 100 cruise missiles into Wuhan and a spare 20 into various Swiss cities, which would end this entire Globonazi / Chinazi farce once and for all.
OK. Save some for North Carolina and Canada, too. It’s complicated.
They COULD make more variants and release them, but nobody wants to screw up and get caught, like they already have been caught, time and time again, to the point that the whole Globonazi plan might finally get run down like a rabid dog in the middle of the road.
The fact of the matter, however, is that even with DRASTIC homing in on Baric, Daszak and Wuhan from the left, with Fauci finally treed by BEAGLES, of all things, and the rest of us bearing down on them from the right and center, they keep pushing on. They are NOT going to stop.
Variants have now died down due to the mechanics of immunity, largely due to refusal of so many people to take the immunosuppressing phony vaccines. But THAT can be worked around. Don’t think that variants are gone. They’re TOO DAMNED USEFUL.
So how do you get MORE of them, without a ChiCom release operation, to convince all the CHUMPS in science, who will swear on their various manuals and codexes that it’s all real?
Just give a CURE that makes sure there are MORE variants.
Remdesivir doesn’t WORK well enough. It makes money, because ALL modern operations have to make their own money, but it doesn’t promote mutation fast enough. Nor is it administered during the viral maximum, when maximum mutation is possible.
Enter molnupiravir.
Move variants needed? Sure! And in time for their NEXT political operation, a.k.a. the 2022 election!
The way this scam of vaccines and drugs works is really smart.
The narrow vaccines NARROW humanity’s pool of immunity coverage of the spike protein, while decreasing overall immunity, both broad-based immunity to COVID and to other diseases. Meanwhile, the drugs WIDEN the shotgun pattern of the spike to find new variants that evade the vaccines.
This is such an incredibly slick grift, I almost have to applaud it. BRAVO! Satan himself has to be IMPRESSED. New diseases hidden in cures for old ones. And all of it helping to achieve the socialist goal of transforming mankind PER SE.
Before this is over, as they begin to move the increasingly narrow coverage genetically, even the original Wuhan strain will become a “variant of concern” for vaxxies! Ah, what a beautiful SCAM. The irony!
Note that this explains why HCQ and ivermectin cannot be used. They dead-end the scam. One has to have something that completes the “scam cycle” of increasing the problem while pretending to fix it.
This is their modus operandi. They find something that looks like solving a problem, that actually perpetuates the problem, or creates a new and similar one.
Just like “pursue gain of function to prevent gain of function” – which scam was revealed by Judy Mikovits.
If you find Democrats like Fauci anywhere NEAR one of these cyclic grifts, you know you’ve identified a scam correctly.
One of the really big shockers for me has been the relationship of the tech giants to the bat coronavirus researchers. What the heck is up with that?
Why did Zuckerberg and Chan (after COVID “struck”) fund Baric, who worked with “the bat woman” and Wuhan? It smells every bit like a “book deal” – an influence pay-off after the fact. This would lead me to believe that Zuck and Chan and Baric actually go back FURTHER, in ways that have not appeared yet.
Why did Google fund Daszak and a group that then funded Wuhan? This actually goes back. And then, Daszak trying to cover up HIS role in the cover-up of the Wuhan Institute of Virology connection – what’s up with THAT?
I don’t want to get into all that stuff too much, but I want to leave you some great links to show you this is REAL – this is not BS. The “fake news snopesing complex” is all over this, trying to hide it, but the word is OUT.
TECH and BAT RESEARCH are THICK.
References on tech / bat research monetary links
Here is the Zuckerberg-Chan “book deal payoff” to Baric’s lab.
Fauci reveals that Mark Zuckerberg offered him “resources and money” – this at the same time Zuck was using cash to interfere in the 2020 election on behalf of Democrats.
Now, I had just heard about all of these monetary connections now being discovered, and thought that any relationship between the tech giants and these virus creeps had to be connected to the FAKE ELECTION PLOT.
BUT – as I was perusing the internet COVID literature, I encountered two scientific papers that made me realize something.
These tech tyrants may have had ANOTHER motive. A DEEPER and MORE PERSONAL motive.
Wanting to live forever – or at least LONGER.
And it gets BIGGER – as in all of society – as in FITNESS – as in EUGENICS.
Here is the part of this paper that set off the alarms. I will make BOLD or underline the most important parts:
BAT MICROBES CAN SHED LIGHT ON DISEASE, IMMUNITY, AND LONGEVITY
Bats and their microbes are increasingly recognized as important components of zoonotic disease cycles (37, 38). A few studies have identified potentially pathogenic members of the excreted bat microbiome such as Bartonella spp. (39, 40) and Leptospira spp. (41). Bats are also known or suspected to be the reservoir of several viruses that are lethal to humans, such as severe acute respiratory syndrome (SARS), Ebola, and rabies viruses (42,–44), as well as of Plasmodium parasites closely related to those in rodents that are used as models to study malaria (45). Genomic insights have generated plausible explanations for how bats may have evolved to harbor such deadly microbes (e.g., reference 46), but in spite of abundant evidence that the microbiome interfaces directly with the host immune system (47, 48), there has not yet been an integrative study addressing whether microbial symbionts contribute to bats’ innate ability to act as pathogen reservoirs. As an additional axis of variation, bats which have flexible roosting habits can be found in close proximity to humans and may potentially swap microbes with humans and their companion animals (49). Bats may transfer microbes to livestock when they exist in close proximity (e.g., pigs consuming partially eaten fruits dropped by fruit bats [50]) or use the same habitats (e.g., horses coming into contact with bat droppings in pastures [51]). Studying bat microbiomes would therefore have obvious public health implications and could help to explain the epidemiology of emerging infectious diseases.
Similar avenues of research can also consider what impact, if any, the host microbiome has on susceptibility of bats to white nose syndrome (WNS), a frequently fatal cutaneous infection that has reduced hibernating bat populations by up to 90% in North America (52). Because not all individuals are killed by the infection, there may be selection on the skin microbiomes of surviving individuals to become enriched with antifungal bacteria. Indeed, one study discovered that in WNS-positive populations, the skin microbiome of bats was enriched with Rhodococcus and Pseudomonas spp., which are known to have antifungal activity (53). Additional studies in this area can answer the questions of how exactly these bacteria inhibit the growth of the causative agent of WNS and what enrichment of the microbiome with these bacteria might mean for the long-term survival of affected host populations.
Bat microbiomes can be used more generally to understand the links between the microbiome and the evolution of other phenomena of interest, such as immunity and longevity. To date, studies addressing the link between host aging and the microbiome in humans and lab animals have uncovered direct links between microbial metabolic products and life span of the host (54). Bats represent an exciting system in which to test for links between the microbiome and aging because they are exceptionally long-lived for a mammal of their size (55, 56). Mice are conventional model mammals, but the commonly used BALB/c mouse strain has a life span of about a year and half, making studies of longevity in these animals rather short-lived (57). Bats of comparable mass can achieve life spans of up to 40 years, and many are philopatric to particular roosts, making repeat sampling of individuals throughout their lifetimes possible (58, 59). Because these animals’ microbiomes can be sampled nonlethally, they are inherently attractive for such studies (36). However, it is worth noting that these animals are especially sensitive to disturbance during hibernation, so experimental designs should minimize unintended disturbance of roosts, particularly in areas where white nose syndrome has decimated bat populations (60). It may also be possible to keep bats in captive colonies in order to sample them throughout successive years of their lives. Recent evidence suggests that metabolites produced by gut microbes in bats might offset the oxidative damages incurred during active flight, resulting in downstream impacts on aging (61). However, many questions still remain. By what mechanism does the microbiome help to extend life span, and is this pattern consistent across mammals? How does interindividual variation impact the relationships between longevity and microbiome community structure? We believe that studies of bat microbiomes can help to answer these important questions and more.Go to:
FUTURE DIRECTIONS
Bats represent an untapped resource for understanding microbiome evolution in mammals. Because of their exceptional diversity, longevity, and ecological importance, we believe that studies of their microbial symbionts will reveal exciting new roles for microbes in driving host evolution and fitness and may help us to better understand the dynamics of emerging zoonotic pathogens. We provide applications of bat microbiome research in the hopes that more researchers will realize the potential that this system has to offer. Multi-omics approaches can be used to parse apart the contributions of host genome, metagenome, and microbial metabolites to the processes described above, and as the costs of these methods continue to decrease, such studies will only become more feasible. The results of studying bat microbiomes using these approaches will undeniably advance the fields of host-microbe interactions, comparative physiology, and public health.
This then led me to a paper specifically about bats and longevity.
This paper is an absolute GOLD MINE of authoritative scientific optimism about life extension through bats, tying in IMMUNITY and INFLAMMATION. I cannot just pick out some part to highlight – the WHOLE THING is evidence of what people are thinking.
What I have done is grabbed the IMAGES which are the most quickly informing. Not all of them – just some of them. Plus a few selections of text which are equally useful.
Abstract
For centuries, people believed that bats possessed sinister powers. Bats are thought to be ancestral hosts to many deadly viruses affecting humans including Ebola, rabies, and most recently SARS-CoV-2 coronavirus. However, bats themselves tolerate these viruses without ill effects. The second power that bats have is their longevity. Bats live much longer than similar-sized land mammals. Here we review how bats’ ability to control inflammation may be contributing to their longevity. The underlying mechanisms may hold clues to developing new treatments for age-related diseases. Now may be the time to use science to exploit the secret powers of bats for human benefit.
Figure 1 Bats Live Longer Than Similar-Sized Land Mammals
(A) Major lineages of bats.
(B) Relationship between lifespan and body mass in mammals. Bats are indicated by red circles; all other species of mammals are indicated by black circles. The lifespan and body mass data are from Healy et al. (2014).
Treatments Based on Bat Strategies
Bats have evolved multiple mechanisms to suppress inflammation, in particular by dampening nucleic acid sensing pathways. A number of pharmacological interventions targeting nucleic acid sensing pathways had already been developed. Historically, the focus has been on developing activators of these pathways to serve as antiviral or anticancer drugs (reviewed in Vanpouille-Box et al., 2019). However, with the realization that inflammation contributes to a wide range of diseases from autoimmunity to age-related conditions, the interest has shifted to developing antagonists of nucleic acid sensors (Sheridan, 2019). This proved to be a challenging task due to high level of redundancy within nucleic acid sensing pathways and the danger of increasing vulnerability to infections. Here, the information obtained from the studies of bats can assist in drug discovery (Table 1 ).
Future Perspectives
In summary, besides serving as a source of deadly diseases and harboring viruses similar to SARS-CoV-2, which caused the current pandemics, bats have a lot to offer humanity by illuminating the pathways to develop novel therapeutics to treat age-related conditions and promote longevity. Already studies of the altered innate immune responses in bats point to several classes of small molecules, some of which have links to aging. However, we can only see the tip of the iceberg when it comes to understanding how bats deal with inflammation, and clearly more studies are warranted. This is also true for other hallmarks of aging, which are still minimally explored. In addition to finding small molecules targeting specific pathways that can be tested in humans, it will be of interest to engineer specific bat alterations in mice and determine whether this leads to enhanced lifespan and healthspan.
Bats have evolved skewed, and ultimately successful, strategies to experience longer and healthier lives, even if this is a secondary outcome of selection for responses to viral infections and/or the dramatic range of metabolic states that accompany periods of flight and torpor. Humans in the last century have created a lifestyle that has gone bats; we live in high densities and (many of us) travel extensively, enhancing exposure to and spread of pathogens. By embracing “batty” strategies to deal with the challenges that our new lifestyle presents, we may be able to solve what look to be the two biggest medical challenges of the 21st century: the rise of viral pandemics and the ever-increasing prevalence of chronic diseases that all share aging as their biggest risk factor.Go to:
Acknowledgments
This perspective was conceived when the authors were quarantined together for potential exposure to COVID-19. Research in authors’ laboratories is supported by grants from the National Institutes of Health, United States.
Where does this go?
OK? It really helps to skim through that SECOND article, because one can ALMOST see the stuff that would make these TECH TITANS DROOL.
Centuries more of life if we just got PARTIAL success in bringing over some “bat tech” to human life.
And I didn’t really dig here, either. This was just what was floating on TOP of the “bat longevity” literature.
Think how much “good” Zuckerberg, Brin, Gates and these others could do, in a few more centuries, if they just had the elixir vitae that bats seem to promise!
Those who don’t think this is a possible motivation, can look at my previous discussion of “life extension” in another article about the more “fictional” science of adrenochrome, wherein I discussed WHY it’s possible to sell a complete fiction like adrenochrome the drug to both CUSTOMERS and CRITICS. In that discussion, the interest of the tech titans in life extension is both mentioned and referenced.
The Truths, Lies, and Disinformation Surrounding a Mythical yet Very Real Substance There is nothing more intriguing than an enigmatic character who defies political and cultural boundaries to tell great truths, yet mixes in just enough lies, fakery, shiny objects, red herrings, exaggerations and omissions to make himself or herself economically viable solely on their …
In the process of this scientific and social examination of the mythical “adrenochrome”, I looked at the very real concept of “young blood”.
There is a REAL market for any REAL or merely RUMORED rejuvenation.
People will even inject themselves with a deadly POISON to look younger.
Yes. “Beautox” is real. And it “REALLY” works – to make people LOOK younger.
Oh, Mark Zuckerberg – you are looking MAAAAHRRRRVELOUS!
Now, I don’t know WHO or HOW – China or CIA or Obama or DARPA – but I suspect that LIFE EXTENSION is the HOBBY WITH BENEFITS of the elite. And BATS was a natural attractor.
So I suspect that this INFLUENCER – bat research as the path to the elixir vitae – was introduced into the RIGHT CIRCLES by people who understood influence. And HERE WE ARE.
W
PS:
How will China and their puppets study the success of their bioweapon here in the United States, and its effect on voting?
